Biological Responses to Hydrogen Molecule and its Preventive Effects on Inflammatory Diseases.
Ikuroh Ohsawa · Current pharmaceutical design · 2021
Research-use notice
Independent study record
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H2HUBB TAKEAWAY
However, the researchers showed previously that inhalation of H₂ markedly suppresses brain injury induced by focal ischemia-reperfusion by buffering oxidative stress. This source-grounded publication contributes evidence relevant to molecular hydrogen and is retained in a transparent general research category when a more specific study design is not supported by the indexed record.
How Molecular Hydrogen Was Addressed
However, the researchers showed previously that inhalation of H₂ markedly suppresses brain injury induced by focal ischemia-reperfusion by buffering oxidative stress.
What the Publication Reported
Specifically, H₂ administration induces mitochondrial oxidative stress and activates Nrf2; this phenomenon, in which mild mitochondrial stress leaves the cell less susceptible to subsequent perturbations, is called mitohormesis.