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Human clinical evidenceHuman Clinical StudyOther or not reported

Hydrogen-rich saline attenuates chemotherapy-induced ovarian injury via regulation of oxidative stress.

Xiaoyin Meng, Hongguang Chen, Guolin Wang, Yonghao Yu, Keliang Xie · Experimental and therapeutic medicine · 2015

Research-use notice

Independent study record

Each H2HUBB study page organizes source-linked research details for educational use. Interpretation should remain proportional to the study design, population, controls, and limitations.

H2HUBB Research Library branded molecular hydrogen research image
Primary topic Molecular Hydrogen Overview
Evidence type Human clinical evidence
Publication type Human Clinical Study
Hydrogen method Other or not reported

H2HUBB TAKEAWAY

In The aim of the present study was to investigate whether hydrogen-rich saline treatment could attenuate ovarian damage induced by cisplatin, hydrogen-rich saline attenuated the FSH release, elevated the level of E2, improved the development of follicles, and reduced the damage to the ovarian cortex at T1, T2 and T3 in the OI + H₂ rats.

What the Researchers Studied

The aim of the present study was to investigate whether hydrogen-rich saline treatment could attenuate ovarian damage induced by cisplatin.

How the Study Was Conducted

Randomized human clinical trial.

How Molecular Hydrogen Was Used

In the OI and OI + H₂ groups, the rats were administered a dose of cisplatin on the 1st and 7th days. The rats in the Con + H₂ and OI + H₂ groups were intraperitoneally injected with hydrogen-rich saline (10ml/kg body weight) once a day over a 2-week period.

What the Researchers Found

Hydrogen-rich saline attenuated the FSH release, elevated the level of E2, improved the development of follicles, and reduced the damage to the ovarian cortex at T1, T2 and T3 in the OI + H₂ rats.

Authors’ Conclusion

The authors concluded that in conclusion, hydrogen-rich saline exerts a protective effect against cisplatin-induced ovarian injury by reducing MDA and increasing SOD and CAT activity.