Hydrogen Research Study
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Preclinical animal evidenceAnimal studyOther or not reported

HydroMg attenuates doxorubicin-induced myocardial injury via promoting mitochondrial function.

Lu Zhang, Yawei Jin, Yuning Zhang, Mohan Li, Xiong Gao, Bin Ma, Xiaonan Ning, Aijun Sun, Wenjiang Ding, Zhiguang Ding, Xiurui Ma, Xiaolei Sun, Jian An · Medical gas research · 2027

Research-use notice

Independent study record

Each H2HUBB study page organizes source-linked research details for educational use. Interpretation should remain proportional to the study design, population, controls, and limitations.

H2HUBB Research Library branded molecular hydrogen research image
Primary topic Radiation and Chemotherapy Injury
Evidence type Preclinical animal evidence
Publication type Animal study
Hydrogen method Other or not reported

H2HUBB TAKEAWAY

In this animal study, the authors report that In vitro studies showed that 1 μg/mL HydroMg treatment attenuated doxorubicin-induced cardiomyocyte death by inhibiting apoptosis, reducing reactive oxygen species production, and restoring mitochondrial membrane potential.

What the Researchers Studied

Source-reported study description: In contrast to molecular hydrogen's intrinsic limitations, HydroMg's structural optimization shortens gastrointestinal transit, reducing hydrogen dissipation while enhancing cellular bioavailability.

Source-reported study description: However, it remains unclear whether it also provides protection against cardiac injury.

What the Study Found

The authors reported: Here, we investigated the cardioprotective effect of HydroMg, a sustained-release hydrogen donor, against doxorubicin-induced cardiac injury.

What the Findings Mean

The authors concluded/reported: In vitro studies showed that 1 μg/mL HydroMg treatment attenuated doxorubicin-induced cardiomyocyte death by inhibiting apoptosis, reducing reactive oxygen species production, and restoring mitochondrial membrane potential.

Study Details

Publication type: animal_study

Evidence source level: B_ABSTRACT

Original scholarly source: https://pubmed.ncbi.nlm.nih.gov/42734445/