Hydrogen Research Study
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Preclinical animal evidenceAnimal studyOther or not reported

Hydrogen-rich saline ameliorates the severity of l-arginine-induced acute pancreatitis in rats.

Han Chen, Yan Ping Sun, Yang Li, Wen Wu Liu, Hong Gang Xiang, Lie Ying Fan, Qiang Sun, Xin Yun Xu, Jian Mei Cai, Can Ping Ruan, Ning Su, Rong Lin Yan, Xue Jun Sun, Qiang Wang · Biochemical and biophysical research communications · 2010

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Independent study record

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H2HUBB Research Library branded molecular hydrogen research image
Primary topic Molecular Hydrogen Overview
Evidence type Preclinical animal evidence
Publication type Animal study
Hydrogen method Other or not reported

H2HUBB TAKEAWAY

In Sprague-Dawley rats, hydrogen-rich saline treatment significantly attenuated the severity of l-Arg-induced AP by ameliorating the increased serum amylase activity, inhibiting neutrophil infiltration, lipid oxidation and pancreatic tissue edema. These results are preclinical and suggest molecular hydrogen's therapeutic potential in the condition studied. Further human research is needed to establish clinical effectiveness.

What the Researchers Studied

Sprague-Dawley rats

How Molecular Hydrogen Was Used

Hydrogen-rich saline (>0.6mM, 6ml/kg) or saline (6ml/kg) was administered, respectively, via tail vein 15min after each l-Arg administration.

What the Researchers Found

Hydrogen-rich saline treatment significantly attenuated the severity of l-Arg-induced AP by ameliorating the increased serum amylase activity, inhibiting neutrophil infiltration, lipid oxidation and pancreatic tissue edema.

Biological or Mechanistic Findings

These results indicate that hydrogen treatment has a protective effect against AP, and the effect is possibly due to its ability to inhibit oxidative stress, apoptosis, NF-kappaB activation and to promote acinar cell proliferation.

Authors’ Conclusion

The authors concluded that these results indicate that hydrogen treatment has a protective effect against AP, and the effect is possibly due to its ability to inhibit oxidative stress, apoptosis, NF-kappaB activation and to promote acinar cell proliferation. These results are preclinical and suggest molecular hydrogen's therapeutic potential in the condition studied. Further human research is needed to establish clinical effectiveness.